Component in Green Tea Found Helpful in Alzheimer's Disease Prevention
Alzheimer's disease is a brain disorder that gradually affects a person's memory as well as their ability to learn, communicate, and carry out daily activities. Alzheimer's also leads to dementia, which is the destruction of brain cells and is characterized by brain-clogging tangles of proteins called plaques. According to a University of South Florida study, a component of green tea called ECGC (epigallocatechin-3-gallate) may reduce or prevent the formation of the detrimental plaques. ECGC is a known antioxidant. It helps the body fight harmful free radicals and promotes cellular longevity. The study also shows that the process of aging tea leaves tends to decrease the amount of antioxidants and that is the reason why green tea is considered to be more powerful in fighting diseases than other teas; black tea is aged the most and green tea the least. Additional studies on ECGC and Alzheimer's would help to confirm the findings of this study. For more information go to the Journal of Neuroscience, September 2005.
Alzheimer's disease is a brain disorder that gradually affects a person's memory as well as their ability to learn, communicate, and carry out daily activities. Alzheimer's also leads to dementia, which is the destruction of brain cells and is characterized by brain-clogging tangles of proteins called plaques. According to a University of South Florida study, a component of green tea called ECGC (epigallocatechin-3-gallate) may reduce or prevent the formation of the detrimental plaques. ECGC is a known antioxidant. It helps the body fight harmful free radicals and promotes cellular longevity. The study also shows that the process of aging tea leaves tends to decrease the amount of antioxidants and that is the reason why green tea is considered to be more powerful in fighting diseases than other teas; black tea is aged the most and green tea the least. Additional studies on ECGC and Alzheimer's would help to confirm the findings of this study. For more information go to the Journal of Neuroscience, September 2005.